From: The interplay of exercise and green tea: a new road in cancer therapy
Type of cancer | Green tea formulation | Effects | Model | Ref |
---|---|---|---|---|
Melanoma | EGCG | Induced cell death | In vitro, In vivo | [147] |
Prostate cancer | Curcumin + Ginger + GT + chamomile | Increased apoptosis | In Vitro | [148] |
Arctigenin + GT + Quercetin | Highly effective in prostate cancer chemoprevention | In vivo | [149] | |
GT + Quercetin | No liver toxicity was observed | Human | [150] | |
Breast cancer | Curcmin + GT | Inhibited tumor growth | In vitro | [151] |
EGCG | Inhibited tumor growth | In vitro, In vivo | [152] | |
Lung cancer | Niosomes –EGCG | Increased apoptosis | In vitro | [153] |
GT | Not appear to offer protective benefits against lung cancer at a population level | Human | [154] | |
Digestive cancer | GT | - | Human | [133] |
Colorectal cancer | EGCG | Inhibited cell proliferation | In vitro, In vivo | [155] |
EGCG | Increased apoptosis and autophagy | In vitro | [156] | |
pancreatic cancer | GT | Repressed the expression of NF-κB | In slico, in vitro | [157] |
Head and Neck | EGCG | Epigenetically silenced tumor suppressors to inhibit the growth of HNSCC cells | In vitro | [158] |
Liver cancer | GT | An inverse association between cumulative consumption of tea, especially green tea and the risk of primary liver cancer | Human | [159] |
Ovarian cancer | EGCG | Targeted intracellular transducing events that regulate the acquisition of an invasive CSC phenotype. | In vitro | [160] |
Bladder cancer | EGCG | Inhibited proliferation, increased autophagy | In vitro, In vivo | [161] |
Osteosarcoma | EGCG | Reduced the stemness and abate drug-resistance of osteosarcoma cells | In vitro | [162] |